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e.g. @Winn:2003hx for a comprehensive review), in which most of the ISM around the BCG is acquired from its immediate environs. This is corroborated by the absence of metals in the group [@Mateo:1998wv], presumably because the interstellar medium is stripped by ram pressure [@Tonnesen:2010iz]. With all the aforementioned evidence, we conclude that the higher redshift groups are the progenitors of local systems. More and better observations are needed to constrain this scenario.

Looking ahead, there are various avenues to expand our study. On the observational side, we are able to obtain detailed and extensive [ SDSS]{} spectroscopic measurements for the 67,389 [ SDSS]{} groups, which will not only reveal even more details on the ISM state of the group but can also serve as powerful background samples to enhance the [[fibre]{}]{} spectroscopic campaigns in future. On the theoretical side, our treatment of the ISM evolution is limited in the following three aspects.

1. The three free parameters to control the gas consumption, namely the efficiency parameter $f$, the gas consumption time $t_\mathrm{sink}$, and the return parameter $R$, are all based on the cosmological context (see Eqs. \[eq:sink\] and \[eq:return\]). This is for two reasons: firstly, this model is obviously applicable to any such feedback process (see below), and secondly, there is no solid evidence to support the observations that suggest a longer gas consumption time for galaxies at high-z.

2. The ISM evolution is only based on the spheroid-driven wind model, whereas other processes (e.g. the stellar mass-driven outflow, see @Weinmann:2011ej; the UV radiation from star-forming galaxies, @Papovich:2011ju; the momentum-conserving winds, @Steinborn:2011
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Dermatology, edited by world authorities Jean L. Bolognia, MD, Joseph L.. Get regular content updates, download illustrations in JPEG or PPT format, andÂ .Monocytes from patients with rheumatoid arthritis and chronic fatigue syndrome possess high levels of tumor necrosis factor.
We recently found that TNFalpha produced by T cells stimulated with anti-CD3 monoclonal antibody could stimulate B cells to synthesize IgE. To examine whether the same phenomenon occurred in other cells such as monocytes, we studied monocytes obtained from patients with chronic fatigue syndrome and rheumatoid arthritis. The levels of both TNFalpha and its receptors, p55 and p75, were significantly higher in monocytes from patients with chronic fatigue syndrome or rheumatoid arthritis than in those from normal donors. Treatment of the monocytes with anti-

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