Xforce Keygen 32bits Or 64bits [NEW] Version Maya 2013 Portable 🚀
公開日:2022/07/06 / 最終更新日:2022/07/06
Xforce Keygen 32bits Or 64bits Version Maya 2013 Portable
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In many murine models of autoimmune disease, T cells are not required for the development of a destructive autoimmune response. Such models include: 1) T cell depleted athymic nude mice (ATN); 2) T cell receptor (TCR)-transgenic mice expressing TCRs which recognize autoantigen; and 3) T cells that are transgenic for the beta2-microglobulin gene, which are thymus-negative (TN). In all 3 systems, pathogenic T cells are generated in athymic nude mice (ATN) that express functional TCRs and respond to self-Ags in vitro. In the CIA model of human RA, pathogenic CD4+T cells are genetically deficient, but sufficient CD4+T cells are generated to initiate disease. It is unknown, however, whether CD4+T cells are required for disease development in this model. We have demonstrated that pathogenic CD4+T cells are generated in both ATN and TCR-transgenic mice. In TCR-transgenic mice, disease can be initiated by transfer of T cells bearing TCRs which are specific for the carboxy-terminal peptide of type II collagen. In ATN mice, a 10 day course of anti-CD4 Abs is sufficient to prevent the development of disease, whereas protection is only observed with > or =7 days of anti-CD4 Ab treatment. These results demonstrate that CD4+T cells are required for CIA. Further studies with the TCR-transgenic CIA model, in which mice with normal TCR repertoire develop disease, indicate that the final effector and effector memory cells are likely to
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